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The resulting product was used for the in vitro specifically the release of entrapped Etoposide in response to the acidic tumor microenvironment, which improved drug accumulation and efficacy [15]

J Neurochem 64: 19651972

In summary, the GSH-GSSG redox cycle does not exist in isolation, but rather operates as a dynamic equilibrium system that is highly coupled to the NADPH metabolic network

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3.2), contribute to neuroprotective signaling, which include improved mitochondrial function, increased BDNF expression, and activation of anti-inflammatory signaling via GPR41/43 and HDAC inhibition [144]

Lage et al., 2022)